https://doi.org/10.1158/1541-7786.c.6672895.v4
P
Pedro Mota
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Pedro Mota is an academic researcher. The author has contributed to research in topics: Mitochondrial Function and Pathology & Genetics and Neurodevelopmental Disorders & Epigenetics and DNA Methylation. The author has an h-index of 4, co-authored 100 publications.
ORCID: 0000-0002-7874-2974Papers by this author
worksData from Overlaid transcriptome and proteome analyses identify mitotic kinesins as important targets of arylsulfonamide-mediated RBM39 degradation
2024 · 0 citations
Supplementary Figure S2 from Overlaid transcriptome and proteome analyses identify mitotic kinesins as important targets of arylsulfonamide-mediated RBM39 degradation
2024 · 0 citations
https://doi.org/10.1158/1541-7786.27034774
Supplementary Figure S1 from Overlaid transcriptome and proteome analyses identify mitotic kinesins as important targets of arylsulfonamide-mediated RBM39 degradation
2024 · 0 citations
https://doi.org/10.1158/1541-7786.27034777
Supplementary Data from Overlaid transcriptome and proteome analyses identify mitotic kinesins as important targets of arylsulfonamide-mediated RBM39 degradation
2024 · 0 citations
https://doi.org/10.1158/1541-7786.27034786
Supplementary Data Table 2 from Overlaid transcriptome and proteome analyses identify mitotic kinesins as important targets of arylsulfonamide-mediated RBM39 degradation
2024 · 0 citations
https://doi.org/10.1158/1541-7786.27034789
Supplementary Figure S7 from Overlaid transcriptome and proteome analyses identify mitotic kinesins as important targets of arylsulfonamide-mediated RBM39 degradation
2024 · 0 citations
https://doi.org/10.1158/1541-7786.27034759
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