Amyloid-Beta and Phosphorylated Tau Accumulations Cause Abnormalities at Synapses of Alzheimer’s disease Neurons
2016 · Journal of Alzheimer s Disease · 637 citations
https://doi.org/10.3233/jad-160612
P
P. Hemachandra Reddy
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P. Hemachandra Reddy is an academic researcher from Texas Tech University. The author has contributed to research in topics: Alzheimer's disease research and treatments & Mitochondrial Function and Pathology & Cholinesterase and Neurodegenerative Diseases. The author has an h-index of 94, co-authored 295 publications.
ORCID: 0000-0002-9560-9948Papers by this author
worksPolyglutamine-expanded Huntingtin Promotes Sensitization of N-Methyl-d-aspartate Receptors via Post-synaptic Density 95
2001 · Journal of Biological Chemistry · 288 citations
https://doi.org/10.1074/jbc.m103501200
RETRACTED: Hippocampal mutant APP and amyloid beta-induced cognitive decline, dendritic spine loss, defective autophagy, mitophagy and mitochondrial abnormalities in a mouse model of Alzheimer’s disease
2018 · Human Molecular Genetics · 285 citations
https://doi.org/10.1093/hmg/ddy042
Mutant APP and amyloid beta-induced defective autophagy, mitophagy, mitochondrial structural and functional changes and synaptic damage in hippocampal neurons from Alzheimer’s disease
2018 · Human Molecular Genetics · 282 citations
https://doi.org/10.1093/hmg/ddy154
Mutant huntingtin, abnormal mitochondrial dynamics, defective axonal transport of mitochondria, and selective synaptic degeneration in Huntington's disease
2011 · Biochimica et Biophysica Acta (BBA) - Molecular Basis of Disease · 177 citations
https://doi.org/10.1016/j.bbadis.2011.10.016
Are mitophagy enhancers therapeutic targets for Alzheimer’s disease?
2022 · Biomedicine & Pharmacotherapy · 71 citations
https://doi.org/10.1016/j.biopha.2022.112918
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