Physiological measurements with varying Cx36 and Kir6.2<sup>[ΔN30,K185Q]</sup> expression.
2016 · Figshare · 0 citations
https://doi.org/10.1371/journal.pcbi.1005116.g002
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J. Westacott Matthew
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J. Westacott Matthew is an academic researcher. The author has contributed to research in topics: Cardiac electrophysiology and arrhythmias & Ion channel regulation and function & Pancreatic function and diabetes. The author has an h-index of 1, co-authored 10 publications.
Papers by this author
worksCalcium imaging and modeling in islets with varying Cx36 and Kir6.2<sup>[ΔN30,K185Q]</sup> expression.
2016 · Figshare · 0 citations
https://doi.org/10.1371/journal.pcbi.1005116.g001
Simulated dependence of islet [Ca<sup>2+</sup>] on electrical coupling upon reduced K<sub>ATP</sub> ATP sensitivity.
2016 · Figshare · 0 citations
https://doi.org/10.1371/journal.pcbi.1005116.g003
Simulated decline in [Ca<sup>2+</sup>] due to Kir6.2 and SUR1 mutations, and recovery following decreased electrical coupling.
2016 · Figshare · 0 citations
https://doi.org/10.1371/journal.pcbi.1005116.g005
Simulated dependence of [Ca<sup>2+</sup>] on electrical coupling following modulation of other K<sub>ATP</sub> parameters.
2016 · Figshare · 0 citations
https://doi.org/10.1371/journal.pcbi.1005116.g004
Simulated decline in insulin secretion due to Kir6.2 and SUR1 mutations.
2016 · Figshare · 0 citations
https://doi.org/10.1371/journal.pcbi.1005116.g008
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